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Aamin (Atenolol)
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Aamin

Aamin belongs to a group of medicines called beta-blockers. It is used to treat high blood pressure (hypertension), angina (heart-related chest pain), irregular heart rhythms (arrhythmia).

Other names for this medication:
Aamin, Adbeta, Aglodepin, Amchek, Amdinx, Amdip, Amlobeat, Amlocard, Amlosac, Amloten, Amnorm, Amodep, Ampine, Amtas, Amvik, Angitol, Atenij, Atenolol, Atloma, Atwin, Betanol, Biocard, Calbeta, Cardenol, Cardiking, Cardpin, Carvasc, Defidin, Espin, Etotan, Glipin, Hynorm, Intalol, Joglit, Loscard, Olpine, Samstar, Tenofed, Tensicard, Tensimin, Ziblok

Similar Products:
Cialis, Pyridium, Vasotec, Mircette, Vantin, Celebrex, Robaxin, Panadol, Valtrex, Cleocin

Also known as: Atenolol

Description

{name} (atenolol) is a beta-blocker that affects the heart and circulation (blood flow through arteries and veins).

{name} is used to treat angina (chest pain) and hypertension (high blood pressure).

{name} is also used to lower the risk of death after a heart attack.

Dosage

Usual Adult Dose of Aamin for Hypertension:

Initial dose: 50 mg orally once a day Maintenance dose: 50 to 100 mg orally once a day Maximum dose: 100 mg per day Comments: -If desired response not achieved after 1 to 2 weeks, increase to 100 mg may be beneficial. -Doses greater than 100 mg once a day did not result in significant additional antihypertensive effects. Use: For the treatment of hypertension alone or in combination with other antihypertensive agents.

Usual Adult Dose of Aamin for Angina Pectoris Prophylaxis:

Initial dose: 50 mg orally once a day -Increase to 100 mg orally once a day after 1 week if optimal response not achieved Maintenance dose: 50 to 200 mg orally once a day Maximum dose: 200 mg per day Comments: -Some patients may require 200 mg per day to attain optimal effect. Use: For the long-term management of angina pectoris due to coronary atherosclerosis.

Usual Adult Dose for Angina Pectoris:

Initial dose: 50 mg orally once a day -Increase to 100 mg orally once a day after 1 week if optimal response not achieved Maintenance dose: 50 to 200 mg orally once a day Maximum dose: 200 mg per day Comments: -Some patients may require 200 mg per day to attain optimal effect. Use: For the long-term management of angina pectoris due to coronary atherosclerosis.

Usual Adult Dose of Aamin for Myocardial Infarction:

50 mg orally twice a day or 100 mg orally once a day Comments: -If IV beta blockers are contraindicated or inappropriate, oral therapy should continue for at least 7 days post-myocardial infarction (MI). -Treatment with beta blockers post MI should generally continue for 1 to 3 years if there are no contraindications. Use: For the management of hemodynamically stable patients with definite or suspected acute myocardial infarction to reduce cardiovascular mortality.

Usual Geriatric Dose for Hypertension:

Initial dose: Consider reducing the starting dose to 25 mg orally once a day

Detailed Aamin dosage information

Overdose

If you overdose Aamin and you don't feel good you should visit your doctor or health care provider immediately. Symptoms of Aamin are low blood sugar, slow heartbeat, heart failure, slowness, low blood pressure, wheezing.

Storage

Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Aamin are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

Appropriate studies have not been performed on the relationship of age to the effects of atenolol injection in the pediatric population. Safety and efficacy have not been established .

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Virulence genotyping of Escherichia coli isolates from diarrheic and urinary tract infections in relation to phylogeny in southeast of Iran. We conclude that in resting murine endothelial cells, absence of endothelial production of NO by itself does not initiate endothelial cell activation or promote leukocyte-endothelial cell interactions. Antagonistic effects of arachidonic acid and phenylarsine oxide. This should help to shed light on the potential mechanism of CHM to improve the usage and clinical efficacy of CHM. Path analysis using parsimonious models gave evidence of significant biologic and environmental transmission for total cholesterol (genetic and cultural heritabilities were 0.46 and 0.05, respectively), high density lipoprotein cholesterol (0.42, 0.10), triglycerides (0.21, 0.07), and systolic (0.48, 0.04) and diastolic (0.35, 0.05) blood pressure. A new experimental pancreatitis by incomplete closed duodenal loop: the influence of pancreatic microcirculation on the development and progression of induced severe pancreatitis in rats. Vagus nerve stimulation: outcome and predictors of seizure freedom in long-term follow-up.

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High-resolution NMR and computer modeling studies of the cannabimimetic aminoalkylindole prototype WIN-55212-2. CL users of rigid gas-permeable CLs (RGPCL) (group 1) or silicone hydrogel CLs (SiHCL) (group 2) were compared with non-CL-using healthy subjects (group 3). Sport-related eye injuries are a common cause of acute ocular injury. Additionally, both physician quality and Internet health information quality have significant effects on patient-physician concordance, with physician quality exhibiting a much stronger relationship. The attractor of the TCD timeseries shows the entire range of states the cardiovascular system can display.

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Effectiveness of composite bone graft substitute plugs in the treatment of chondral and osteochondral lesions of the talus. Time of acquisition and network stability in pediatric resting-state functional magnetic resonance imaging. Qualitative interviews were conducted with 50 social work practitioners in four Canadian cities to gain insight into methods of practice that are culturally sensitive for work with Muslim clients. Overexpression of the antisense RNA resulted in a significant increase in parC mRNA levels. Therefore deeper understanding of the disease mechanism is necessary to develop novel therapeutic strategies.

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