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Bemet (Metformin)

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Bemet is used to treat high blood sugar levels that are caused by a type of diabetes mellitus or sugar diabetes called type 2 diabetes. With this type of diabetes, insulin produced by the pancreas is not able to get sugar into the cells of the body where it can work properly.

Other names for this medication:
Asoride, Benforce, Conformin, Diafer, Diamin, Diapride, Dokmet, Emfor, Etformin, Formet, Forminal, Getmet, Glimkap, Glinate, Glyciphage, Glyciz, Glyrep, Insumet, Mefnor, Mefomin, Meglipride, Metadoze, Metanorm, Metformin, Metmin, Metneed, Metsafe, Minitide, Mintide, Obimet, Redidrop, Roglin, Rosizon, Serformin, Tenliptin, Volabay, Walaphage, Zoform, Zomelis

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Also known as: Metformin


{name} (metformin) is an oral diabetes medicine that helps control blood sugar levels.

{name} is used together with diet and exercise to improve blood sugar control in adults with type 2 diabetes mellitus.

{name} is sometimes used together with insulin or other medications, but metformin is not for treating type 1 diabetes.


Usual Adult Dose for Diabetes Type 2:

Immediate-release: Initial dose: 500 mg orally twice a day or 850 mg orally once a day Dose titration: Increase in 500 mg weekly increments or 850 mg every 2 weeks as tolerated Maintenance dose: 2000 mg daily in divided doses Maximum dose: 2550 mg/day Extended-release: Initial dose: 500 to 1000 mg orally once a day Dose titration: Increase in 500 mg weekly increments as tolerated Maintenance dose: 2000 mg daily Maximum dose: 2500 mg daily Comments: -Metformin, if not contraindicated, is the preferred initial pharmacologic agent for treatment of type 2 diabetes mellitus. -Immediate-release: Take in divided doses 2 to 3 times a day with meals; titrate slowly to minimize gastrointestinal side effects. In general, significant responses are not observed with doses less than 1500 mg/day. -Extended-release: Take with the evening meal; if glycemic control is not achieved with 2000 mg once a day, may consider 1000 mg of extended-release product twice a day; if glycemic control is still not achieve, may switch to immediate-release product. Use: To improve glycemic control in adults with type 2 diabetes mellitus as an adjunct to diet and exercise.

Usual Pediatric Dose for Diabetes Type 2:

10 years or older: Immediate-release: Initial dose: 500 mg orally twice a day Dose titration: Increase in 500 mg weekly increments as tolerated Maintenance dose: 2000 mg daily Maximum dose: 2000 mg daily Comments: Take in divided doses 2 to 3 times a day with meals. Titrate slowly to minimize gastrointestinal side effects. Safety and effectiveness of Bemet extended-release has not been established in pediatric patients less than 18 years of age. Use: To improve glycemic control in children with type 2 diabetes mellitus as an adjunct to diet and exercise.

Detailed Bemet dosage information


Seek emergency medical attention or call the Poison Help line at 1-800-222-1222. An overdose can cause severe hypoglycemia or lactic acidosis.


Store at room temperature between 15 and 30 degrees C (59 and 86 degrees F) away from moisture, light and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Bemet are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Avoid drinking alcohol. It lowers blood sugar and may increase your risk of lactic acidosis.

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Sodium Channel Nav1.8 Underlies TTX-Resistant Axonal Action Potential Conduction in Somatosensory C-Fibers of Distal Cutaneous Nerves. PLANTAIN AS A WOUND-HEALING DRUG IN POPULAR AND SCIENTIFIC MEDICINE. MECHANISM OF THE CENTRAL STIMULANT ACTION OF ALPHA-METHYL-META-TYROSINE. Invasive species cover and richness were not significantly different across sites. However, the postural adjustment of the ipsilateral forelimb persists, with the same range of latencies as before the operation. Verification of relationship status is an important component of dyadic research. The mechanism by which simple steatosis progresses to steatohepatitis is not entirely clear. Because antibody appeared to be the major blocking factor in serum, a rapid and efficient heat-extraction method for recovering mannan from antibody-containing serum was devised. Inactivation is controlled by inhibitory phosphorylation of the kinase subunit, by ubiquitin-mediated degradation of the cyclin subunit and by interaction of the complex with small inhibitory proteins.

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